Putative adverse outcome pathway development based on physiological responses of female fathead minnows to model estrogen versus androgen receptor agonists
"Morshead ML, Jensen KM, Ankley GT, Vliet S, LaLone CA, Aller AV, Watanabe KH, Villeneuve DL. Putative adverse outcome pathway development based on physiological responses of female fathead minnows to model estrogen versus androgen receptor agonists. Aquat Toxicol. 2023 Aug;261:106607. doi: 10.1016/j.aquatox.2023.106607. Epub 2023 Jun 9. PMID: 37354817.".
This dataset is associated with the following publication:
Morshead, M., K. Jensen, G. Ankley, S. Vliet, C. LaLone, D. Villeneuve, A. Vidales Aller, and K. Watanabe. Putative adverse outcome pathway development based on physiological responses of female fathead minnows to model estrogen versus androgen receptor agonists. AQUATIC TOXICOLOGY. Elsevier Science Ltd, New York, NY, USA, 261: 106607, (2023).
Complete Metadata
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"fn": "Daniel Villeneuve",
"hasEmail": "mailto:villeneuve.dan@epa.gov"
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| description | "Morshead ML, Jensen KM, Ankley GT, Vliet S, LaLone CA, Aller AV, Watanabe KH, Villeneuve DL. Putative adverse outcome pathway development based on physiological responses of female fathead minnows to model estrogen versus androgen receptor agonists. Aquat Toxicol. 2023 Aug;261:106607. doi: 10.1016/j.aquatox.2023.106607. Epub 2023 Jun 9. PMID: 37354817.". This dataset is associated with the following publication: Morshead, M., K. Jensen, G. Ankley, S. Vliet, C. LaLone, D. Villeneuve, A. Vidales Aller, and K. Watanabe. Putative adverse outcome pathway development based on physiological responses of female fathead minnows to model estrogen versus androgen receptor agonists. AQUATIC TOXICOLOGY. Elsevier Science Ltd, New York, NY, USA, 261: 106607, (2023). |
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| license | https://pasteur.epa.gov/license/sciencehub-license.html |
| modified | 2023-07-07 |
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| references |
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| rights |
null
|
| title | Putative adverse outcome pathway development based on physiological responses of female fathead minnows to model estrogen versus androgen receptor agonists |
